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Creators/Authors contains: "Wang, Xiaoqi"

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  1. ; ; ; ; (, NeurIPS)
    Free, publicly-accessible full text available December 12, 2025
  2. ; ; ; ; ; ; ; ; ; ; et al (, IEEE Computer Graphics and Applications)
    Full Text Available 
  3. ; ; ; ; ; ; ; ; ; ; et al (, Frontiers in Cellular Neuroscience)
    null (Ed.)
    Although the increased expression of members of the chondroitin sulfate proteoglycan family, such as neuron-glial antigen 2 (NG2), have been well documented after an injury to the spinal cord, a complete picture as to the cellular origins and function of this NG2 expression has yet to be made. Using a spinal cord injury (SCI) mouse model, we describe that some infiltrated bone marrow-derived macrophages (BMDMΦ) are early contributors to NG2/CSPG4 expression and secretion after SCI. We demonstrate for the first time that a lesion-related form of cellular debris generated from damaged myelin sheaths can increase NG2/CSPG4 expression in BMDMΦ, which then exhibit enhanced proliferation and decreased phagocytic capacity. These results suggest that BMDMΦ may play a much more nuanced role in secondary spinal cord injury than previously thought, including acting as early contributors to the NG2 component of the glial scar. 
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